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precede the acetyl group transfer. The highly conserved Tyr441 located in the water channel that connects to the a-amino group (see previous section), is positioned to play a role as the catalytic base in proton removal. The lower activity of Y441F mutant is consistent with this catalytic role of this tyrosine. The 7 fold lower activity for N479A mutant confirmed that it is a key residue to bind L
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ree GWAS [9?11]. Significant eQTLs in this gene only replicated in UBC, but the same direction of effect was also observed in the Goningen set. These results supported that HHIP is the most likely causal gene in the region. There are many genes present in the 19q13 locus. This locus was recently associated with COPD and so far no replication study has been published [11]. 222 eQTLs were detected i
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